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Ce produit n'est pas destiné à diagnostiquer, traiter, guérir ou prévenir toute maladie. Ces déclarations n'ont pas été évaluées par la Food and Drug Administration.
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Ces informations sont fournies à titre éducatif uniquement et ne remplacent pas un avis médical professionnel, un diagnostic ou un traitement. Consultez toujours votre professionnel de santé avant d'utiliser des plantes, surtout si vous êtes enceinte, allaitez, prenez des médicaments ou avez une condition médicale.
Anticlea venenosus
Highly toxic North American plant; fatal if confused with edible camas; no medicinal use.
Zigadenus venenosus, commonly known as death camas, is a highly toxic perennial herb native to western North America. It contains steroidal alkaloids such as zigacine and veratrum-type compounds that cause severe cardiac and neurological toxicity. There is no recognized medicinal use; the plant is primarily of concern for accidental poisoning, often due to confusion with edible camas (Camassia spp.).
The primary toxic alkaloids in Zigadenus venenosus (e.g., zigacine, zygadenine) act as sodium channel activators, prolonging depolarization and increasing intracellular calcium in cardiac and neural tissues. This leads to bradycardia, hypotension, and eventually cardiac arrest. Additionally, veratrum-like alkaloids stimulate vagal afferents, inducing the Bezold-Jarisch reflex, which exacerbates hypotension and bradycardia. The compounds also inhibit acetylcholinesterase, contributing to cholinergic crisis symptoms such as salivation, lacrimation, and gastrointestinal distress.
Highly toxic North American plant; fatal if confused with edible camas; no medicinal use.
Zigadenus venenosus, commonly known as death camas, is a highly toxic perennial herb native to western North America. It contains steroidal alkaloids such as zigacine and veratrum-type compounds that cause severe cardiac and neurological toxicity. There is no recognized medicinal use; the plant is primarily of concern for accidental poisoning, often due to confusion with edible camas (Camassia spp.).
The primary toxic alkaloids in Zigadenus venenosus (e.g., zigacine, zygadenine) act as sodium channel activators, prolonging depolarization and increasing intracellular calcium in cardiac and neural tissues. This leads to bradycardia, hypotension, and eventually cardiac arrest. Additionally, veratrum-like alkaloids stimulate vagal afferents, inducing the Bezold-Jarisch reflex, which exacerbates hypotension and bradycardia. The compounds also inhibit acetylcholinesterase, contributing to cholinergic crisis symptoms such as salivation, lacrimation, and gastrointestinal distress.