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Ce produit n'est pas destiné à diagnostiquer, traiter, guérir ou prévenir toute maladie. Ces déclarations n'ont pas été évaluées par la Food and Drug Administration.
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Ces informations sont fournies à titre éducatif uniquement et ne remplacent pas un avis médical professionnel, un diagnostic ou un traitement. Consultez toujours votre professionnel de santé avant d'utiliser des plantes, surtout si vous êtes enceinte, allaitez, prenez des médicaments ou avez une condition médicale.
Zornia latifolia
Tropical American herb used in folk medicine as fever remedy and analgesic.
Zornia latifolia is a tropical herb traditionally used in South American folk medicine for fever and pain. Modern research suggests antipyretic and analgesic properties, attributed to flavonoids, alkaloids, and tannins. Evidence is limited (Level C), primarily from preclinical studies.
The antipyretic effect is thought to involve inhibition of cyclooxygenase (COX) enzymes, reducing prostaglandin synthesis in the hypothalamus. Analgesic activity may be mediated through peripheral opioid receptors and modulation of transient receptor potential (TRP) channels. Flavonoids such as quercetin and kaempferol contribute to anti-inflammatory actions by suppressing NF-κB and COX-2 expression. Alkaloids may interact with central nervous system receptors, though specific pathways remain unclear.
Tropical American herb used in folk medicine as fever remedy and analgesic.
Zornia latifolia is a tropical herb traditionally used in South American folk medicine for fever and pain. Modern research suggests antipyretic and analgesic properties, attributed to flavonoids, alkaloids, and tannins. Evidence is limited (Level C), primarily from preclinical studies.
The antipyretic effect is thought to involve inhibition of cyclooxygenase (COX) enzymes, reducing prostaglandin synthesis in the hypothalamus. Analgesic activity may be mediated through peripheral opioid receptors and modulation of transient receptor potential (TRP) channels. Flavonoids such as quercetin and kaempferol contribute to anti-inflammatory actions by suppressing NF-κB and COX-2 expression. Alkaloids may interact with central nervous system receptors, though specific pathways remain unclear.