PubMed-compiled information sheet
This sheet was compiled from PubMed (NIH) abstracts using AI assistance. Every factual claim is cited to a real PubMed article (see the source list). It has not yet been human-reviewed — confirm with a healthcare provider before use.
Compiled from 30 PubMed articles · model: gemma4:31b
Summary
Background
Traditional uses
Active compounds
Mechanism of action
Clinical evidence
Masson pine pollen aqueous extract (MPPAE) ameliorated kidney damage in rats by altering superoxide dismutase (SOD) and malondialdehyde (MDA) concentrations [PMID:38143799]
PPPS mitigated myocardial tissue injury and suppressed the inflammatory response in animal models and protected H9c2 cells in vitro [PMID:39551317]
Pine pollen polysaccharides (PPM60-III) and sulfated polysaccharides (SPPM60-III) increased body weight, decreased DAI scores, and reduced pro-inflammatory factors in mice [PMID:36431783]
PPPS accelerated wound healing in mouse cutaneous wounds and impaired high glucose-induced apoptosis and migration repression of fibroblasts, keratinocytes, and HUVECs [PMID:40647215]
Pine pollen polysaccharides (PPPS) were identified as the principal bioactive constituent with protective efficacy against DNCB-induced damage in HaCaT cells and BALB/c mice [PMID:40871078]
Safety & adverse effects
Evidence summary
PubMed sources
- 1.PMID: 38331121 (2024) — Masson Pine pollen (Pinus massoniana) activate HD11 chicken macrophages invitro. · Journal of ethnopharmacology
- 2.PMID: 34327226 (2020) — The Potential Effects and Use of Chinese Herbal Medicine Pine Pollen (Pinus pollen): A Bibliometric Analysis of Pharmacological and Clinical Studies. · World journal of traditional Chinese medicine
- 3.PMID: 3310401 (1987) — Pathogenesis of sarcoidosis. · The Western journal of medicine
- 4.PMID: 27852122 (2016) — Pinus massoniana Bark Extract: Structure-Activity Relationship and Biomedical Potentials. · The American journal of Chinese medicine
- 5.PMID: 38143799 (2023) — Masson pine pollen aqueous extract ameliorates cadmium-induced kidney damage in rats.