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Ce produit n'est pas destiné à diagnostiquer, traiter, guérir ou prévenir toute maladie. Ces déclarations n'ont pas été évaluées par la Food and Drug Administration.
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Ces informations sont fournies à titre éducatif uniquement et ne remplacent pas un avis médical professionnel, un diagnostic ou un traitement. Consultez toujours votre professionnel de santé avant d'utiliser des plantes, surtout si vous êtes enceinte, allaitez, prenez des médicaments ou avez une condition médicale.
Cirsium japonicum
Chardon médicinal d'Extrême-Orient utilisé en médecine traditionnelle chinoise pour l'hémostase et les états de stase sanguine.
Cirsium japonicum, an East Asian medicinal thistle, is traditionally used in TCM for hemostasis and blood stasis conditions. Modern research indicates potential hemostatic, anti-inflammatory, hepatoprotective, and antihypertensive effects. Key active compounds include pectolinarin, luteolin, apigenin, linarin, stigmasterol, and beta-sitosterol.
The hemostatic action of Cirsium japonicum is attributed to flavonoids such as luteolin and apigenin, which promote platelet aggregation and vasoconstriction via thromboxane A2 receptor activation. Anti-inflammatory effects are mediated through inhibition of COX-2 and 5-LOX, reducing prostaglandin and leukotriene synthesis. Hepatoprotective properties involve antioxidant activity via Nrf2 pathway activation and suppression of CYP2E1. Antihypertensive effects may result from calcium channel blockade and ACE inhibition, leading to vasodilation.
Chardon médicinal d'Extrême-Orient utilisé en médecine traditionnelle chinoise pour l'hémostase et les états de stase sanguine.
Cirsium japonicum, an East Asian medicinal thistle, is traditionally used in TCM for hemostasis and blood stasis conditions. Modern research indicates potential hemostatic, anti-inflammatory, hepatoprotective, and antihypertensive effects. Key active compounds include pectolinarin, luteolin, apigenin, linarin, stigmasterol, and beta-sitosterol.
The hemostatic action of Cirsium japonicum is attributed to flavonoids such as luteolin and apigenin, which promote platelet aggregation and vasoconstriction via thromboxane A2 receptor activation. Anti-inflammatory effects are mediated through inhibition of COX-2 and 5-LOX, reducing prostaglandin and leukotriene synthesis. Hepatoprotective properties involve antioxidant activity via Nrf2 pathway activation and suppression of CYP2E1. Antihypertensive effects may result from calcium channel blockade and ACE inhibition, leading to vasodilation.