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Ce produit n'est pas destiné à diagnostiquer, traiter, guérir ou prévenir toute maladie. Ces déclarations n'ont pas été évaluées par la Food and Drug Administration.
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Ces informations sont fournies à titre éducatif uniquement et ne remplacent pas un avis médical professionnel, un diagnostic ou un traitement. Consultez toujours votre professionnel de santé avant d'utiliser des plantes, surtout si vous êtes enceinte, allaitez, prenez des médicaments ou avez une condition médicale.
Pueraria lobata
Root of kudzu vine used in TCM for muscle stiffness, fever, diabetes, and now studied for alcohol addiction and cardiovascular conditions.
Pueraria lobata (kudzu) root, known as Ge Gen in Traditional Chinese Medicine, is used for cardiovascular protection, alcohol craving reduction, and menopausal symptom relief. Its primary active compounds include puerarin, daidzin, daidzein, and genistein, which exert estrogenic, anti-inflammatory, and metabolic effects.
Puerarin acts as a selective estrogen receptor modulator (SERM) and inhibits platelet aggregation via cAMP elevation. Daidzin suppresses alcohol intake by inhibiting aldehyde dehydrogenase (ALDH2), leading to aversive acetaldehyde accumulation. Isoflavones also reduce oxidative stress through Nrf2 pathway activation and downregulate NF-κB-mediated inflammation.
Root of kudzu vine used in TCM for muscle stiffness, fever, diabetes, and now studied for alcohol addiction and cardiovascular conditions.
Pueraria lobata (kudzu) root, known as Ge Gen in Traditional Chinese Medicine, is used for cardiovascular protection, alcohol craving reduction, and menopausal symptom relief. Its primary active compounds include puerarin, daidzin, daidzein, and genistein, which exert estrogenic, anti-inflammatory, and metabolic effects.
Puerarin acts as a selective estrogen receptor modulator (SERM) and inhibits platelet aggregation via cAMP elevation. Daidzin suppresses alcohol intake by inhibiting aldehyde dehydrogenase (ALDH2), leading to aversive acetaldehyde accumulation. Isoflavones also reduce oxidative stress through Nrf2 pathway activation and downregulate NF-κB-mediated inflammation.