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Taxillus chinensis
A TCM herb derived from mistletoe parasitizing mulberry trees; tonifies liver and kidney, strengthens bones, and calms the fetus during threatened miscarriage.
Loranthus (Taxillus chinensis) is a traditional Chinese medicinal herb used to tonify the liver and kidneys, strengthen bones, and calm the fetus. It contains active compounds such as quercetin, avicularin, hyperoside, and myricetin, which contribute to its anti-rheumatic, hypotensive, and uterine-relaxant effects. Modern research supports its use for chronic joint conditions, blood pressure support, and threatened miscarriage.
The herb's anti-rheumatic effects are attributed to flavonoids like quercetin and hyperoside, which inhibit cyclooxygenase-2 (COX-2) and reduce pro-inflammatory cytokines. Its hypotensive action may involve angiotensin-converting enzyme (ACE) inhibition and nitric oxide-mediated vasodilation. For fetal stabilization, compounds such as avicularin are thought to relax uterine smooth muscle via beta-adrenergic receptor activation and calcium channel modulation.
A TCM herb derived from mistletoe parasitizing mulberry trees; tonifies liver and kidney, strengthens bones, and calms the fetus during threatened miscarriage.
Loranthus (Taxillus chinensis) is a traditional Chinese medicinal herb used to tonify the liver and kidneys, strengthen bones, and calm the fetus. It contains active compounds such as quercetin, avicularin, hyperoside, and myricetin, which contribute to its anti-rheumatic, hypotensive, and uterine-relaxant effects. Modern research supports its use for chronic joint conditions, blood pressure support, and threatened miscarriage.
The herb's anti-rheumatic effects are attributed to flavonoids like quercetin and hyperoside, which inhibit cyclooxygenase-2 (COX-2) and reduce pro-inflammatory cytokines. Its hypotensive action may involve angiotensin-converting enzyme (ACE) inhibition and nitric oxide-mediated vasodilation. For fetal stabilization, compounds such as avicularin are thought to relax uterine smooth muscle via beta-adrenergic receptor activation and calcium channel modulation.