PubMed-compiled information sheet
This sheet was compiled from PubMed (NIH) abstracts using AI assistance. Every factual claim is cited to a real PubMed article (see the source list). It has not yet been human-reviewed — confirm with a healthcare provider before use.
Compiled from 30 PubMed articles · model: gemma4:31b
Summary
Background
Traditional uses
Active compounds
Mechanism of action
Clinical evidence
A white tea extract mouthrinse potently inhibits plaque formation, though it is not as comparable to chlorhexidine [PMID: 29242687].
White tea intake increased serum antioxidant capacity in endurance-trained rats in a dose-dependent manner and downregulated inflammatory markers IL-1β and IL-6 [PMID: 35800140].
A triple-helix white tea polysaccharide (WTP-2) alleviated dextran sulfate sodium-induced colitis, reducing disease activity index scores by 54.5% [PMID: 41371855].
White tea markedly accelerated re-entrainment to 6-hour phase advances in mice [PMID: 41315249].
White tea extract inhibits maltase and glucoamylase activity in rat intestinal extracts, and decreases starch digestion [PMID: 34029903, 34865796].
Safety & adverse effects
Pregnancy & lactation
Evidence summary
PubMed sources
- 1.PMID: 38056775 (2024) — Biological potential and mechanisms of Tea's bioactive compounds: An Updated review. · Journal of advanced research
- 2.PMID: 34029903 (2021) — Inhibition of α-glucosidases by tea polyphenols in rat intestinal extract and Caco-2 cells grown on Transwell. · Food chemistry
- 3.PMID: 27618035 (2016) — Polyphenols and Sunburn. · International journal of molecular sciences
- 4.PMID: 26978340 (2016) — Recent Advances in Volatiles of Teas. · Molecules (Basel, Switzerland)
- 5.PMID: 34865796 (2021) — The inhibitory action of purple tea on in vivo starch digestion compared to other Camellia sinensis teas.