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Ce produit n'est pas destiné à diagnostiquer, traiter, guérir ou prévenir toute maladie. Ces déclarations n'ont pas été évaluées par la Food and Drug Administration.
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Ces informations sont fournies à titre éducatif uniquement et ne remplacent pas un avis médical professionnel, un diagnostic ou un traitement. Consultez toujours votre professionnel de santé avant d'utiliser des plantes, surtout si vous êtes enceinte, allaitez, prenez des médicaments ou avez une condition médicale.
Aethusa cynapium
Apiaceae européenne toxique pour l'homme ; mentionnée pour la sensibilisation à la sécurité car elle ressemble au persil.
Aethusa cynapium, commonly known as fool's parsley, is a highly toxic plant from the Apiaceae family often mistaken for edible parsley. It contains neurotoxic alkaloids including cynapine and coniine analogs, and has no recognized medicinal or culinary use due to its severe toxicity. Its primary relevance is in toxicology and poisoning prevention.
The principal toxic alkaloids in Aethusa cynapium, such as cynapine and coniine-like compounds, act as nicotinic acetylcholine receptor antagonists, leading to neuromuscular blockade and central nervous system depression. These compounds also exhibit parasympatholytic effects, contributing to symptoms like mydriasis, tachycardia, and respiratory paralysis. The plant's toxicity is dose-dependent, with severe poisoning potentially fatal due to respiratory failure.
Apiaceae européenne toxique pour l'homme ; mentionnée pour la sensibilisation à la sécurité car elle ressemble au persil.
Aethusa cynapium, commonly known as fool's parsley, is a highly toxic plant from the Apiaceae family often mistaken for edible parsley. It contains neurotoxic alkaloids including cynapine and coniine analogs, and has no recognized medicinal or culinary use due to its severe toxicity. Its primary relevance is in toxicology and poisoning prevention.
The principal toxic alkaloids in Aethusa cynapium, such as cynapine and coniine-like compounds, act as nicotinic acetylcholine receptor antagonists, leading to neuromuscular blockade and central nervous system depression. These compounds also exhibit parasympatholytic effects, contributing to symptoms like mydriasis, tachycardia, and respiratory paralysis. The plant's toxicity is dose-dependent, with severe poisoning potentially fatal due to respiratory failure.