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Ce produit n'est pas destiné à diagnostiquer, traiter, guérir ou prévenir toute maladie. Ces déclarations n'ont pas été évaluées par la Food and Drug Administration.
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Ces informations sont fournies à titre éducatif uniquement et ne remplacent pas un avis médical professionnel, un diagnostic ou un traitement. Consultez toujours votre professionnel de santé avant d'utiliser des plantes, surtout si vous êtes enceinte, allaitez, prenez des médicaments ou avez une condition médicale.
Convallaria majalis
Fleur européenne printanière appréciée aux glycosides cardiaques puissants utilisés en médecine populaire et en pharmacie pour les affections cardiaques.
Convallaria majalis (Lily of the Valley) is a cardiotonic herb containing cardiac glycosides such as convallatoxin and convalloside. It has been used traditionally for heart failure and edema, but due to its narrow therapeutic index and high toxicity, modern use is restricted to pharmaceutical preparations under strict medical supervision.
The primary mechanism involves inhibition of the Na+/K+-ATPase pump in cardiac myocytes, leading to increased intracellular sodium and subsequent calcium influx via the Na+/Ca2+ exchanger. This enhances myocardial contractility (positive inotropy). Additionally, the glycosides exhibit vagomimetic effects, slowing heart rate and improving diastolic function. The diuretic action is attributed to increased renal blood flow and inhibition of tubular reabsorption.
Fleur européenne printanière appréciée aux glycosides cardiaques puissants utilisés en médecine populaire et en pharmacie pour les affections cardiaques.
Convallaria majalis (Lily of the Valley) is a cardiotonic herb containing cardiac glycosides such as convallatoxin and convalloside. It has been used traditionally for heart failure and edema, but due to its narrow therapeutic index and high toxicity, modern use is restricted to pharmaceutical preparations under strict medical supervision.
The primary mechanism involves inhibition of the Na+/K+-ATPase pump in cardiac myocytes, leading to increased intracellular sodium and subsequent calcium influx via the Na+/Ca2+ exchanger. This enhances myocardial contractility (positive inotropy). Additionally, the glycosides exhibit vagomimetic effects, slowing heart rate and improving diastolic function. The diuretic action is attributed to increased renal blood flow and inhibition of tubular reabsorption.