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Ce produit n'est pas destiné à diagnostiquer, traiter, guérir ou prévenir toute maladie. Ces déclarations n'ont pas été évaluées par la Food and Drug Administration.
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Ces informations sont fournies à titre éducatif uniquement et ne remplacent pas un avis médical professionnel, un diagnostic ou un traitement. Consultez toujours votre professionnel de santé avant d'utiliser des plantes, surtout si vous êtes enceinte, allaitez, prenez des médicaments ou avez une condition médicale.
Crataegus oxyacantha
Full spectrum hawthorn product combining leaves, flowers, and berries for maximum cardiovascular benefit.
Hawthorn (Crataegus oxyacantha) is a full-spectrum cardiovascular herb whose leaves, flowers, and berries provide synergistic support for heart function. Its primary active compounds—oligomeric proanthocyanidins, hyperoside, vitexin-rhamnoside, chlorogenic acid, and quercetin—contribute to improved coronary blood flow, blood pressure regulation, and antioxidant protection. Evidence level A supports its use in cardiac insufficiency, hypertension, and atherosclerosis prevention.
Hawthorn enhances myocardial contractility and coronary vasodilation primarily through inhibition of phosphodiesterase (PDE) and modulation of calcium channels, increasing intracellular cAMP and improving cardiac output. Its oligomeric proanthocyanidins (OPCs) and flavonoids (e.g., hyperoside, vitexin-rhamnoside) exert positive inotropic effects by sensitizing cardiac myofilaments to calcium and prolonging the action potential. Additionally, hawthorn reduces peripheral vascular resistance via endothelial nitric oxide synthase (eNOS) activation, leading to vasodilation and blood pressure reduction. The herb also demonstrates antioxidant and anti-inflammatory actions by scavenging free radicals and inhibiting NF-κB, which may slow atherosclerotic progression.
Full spectrum hawthorn product combining leaves, flowers, and berries for maximum cardiovascular benefit.
Hawthorn (Crataegus oxyacantha) is a full-spectrum cardiovascular herb whose leaves, flowers, and berries provide synergistic support for heart function. Its primary active compounds—oligomeric proanthocyanidins, hyperoside, vitexin-rhamnoside, chlorogenic acid, and quercetin—contribute to improved coronary blood flow, blood pressure regulation, and antioxidant protection. Evidence level A supports its use in cardiac insufficiency, hypertension, and atherosclerosis prevention.
Hawthorn enhances myocardial contractility and coronary vasodilation primarily through inhibition of phosphodiesterase (PDE) and modulation of calcium channels, increasing intracellular cAMP and improving cardiac output. Its oligomeric proanthocyanidins (OPCs) and flavonoids (e.g., hyperoside, vitexin-rhamnoside) exert positive inotropic effects by sensitizing cardiac myofilaments to calcium and prolonging the action potential. Additionally, hawthorn reduces peripheral vascular resistance via endothelial nitric oxide synthase (eNOS) activation, leading to vasodilation and blood pressure reduction. The herb also demonstrates antioxidant and anti-inflammatory actions by scavenging free radicals and inhibiting NF-κB, which may slow atherosclerotic progression.