Loading...
Ce produit n'est pas destiné à diagnostiquer, traiter, guérir ou prévenir toute maladie. Ces déclarations n'ont pas été évaluées par la Food and Drug Administration.
Loading...
Ces informations sont fournies à titre éducatif uniquement et ne remplacent pas un avis médical professionnel, un diagnostic ou un traitement. Consultez toujours votre professionnel de santé avant d'utiliser des plantes, surtout si vous êtes enceinte, allaitez, prenez des médicaments ou avez une condition médicale.
Magnolia officinalis
TCM herb that moves qi, dries dampness, and directs rebellious qi downward. Used for digestive stagnation, phlegm obstruction, and anxiety. Contains magnolol and honokiol.
Magnolia officinalis bark is a traditional Chinese herb used to move qi, dry dampness, and direct rebellious qi downward. Its primary active compounds, magnolol and honokiol, exhibit anxiolytic, anti-inflammatory, and antimicrobial properties. Modern research supports its use for anxiety, digestive discomfort, and as an adjunct in inflammatory conditions.
Magnolol and honokiol modulate GABA-A receptors, enhancing chloride ion conductance to produce anxiolytic effects. They inhibit COX-2 and NF-κB pathways, reducing pro-inflammatory cytokines. Additionally, these compounds exert antimicrobial activity by disrupting bacterial cell membranes and inhibiting efflux pumps. In the gastrointestinal tract, they promote motility via 5-HT4 receptor agonism and inhibit gastric acid secretion.
TCM herb that moves qi, dries dampness, and directs rebellious qi downward. Used for digestive stagnation, phlegm obstruction, and anxiety. Contains magnolol and honokiol.
Magnolia officinalis bark is a traditional Chinese herb used to move qi, dry dampness, and direct rebellious qi downward. Its primary active compounds, magnolol and honokiol, exhibit anxiolytic, anti-inflammatory, and antimicrobial properties. Modern research supports its use for anxiety, digestive discomfort, and as an adjunct in inflammatory conditions.
Magnolol and honokiol modulate GABA-A receptors, enhancing chloride ion conductance to produce anxiolytic effects. They inhibit COX-2 and NF-κB pathways, reducing pro-inflammatory cytokines. Additionally, these compounds exert antimicrobial activity by disrupting bacterial cell membranes and inhibiting efflux pumps. In the gastrointestinal tract, they promote motility via 5-HT4 receptor agonism and inhibit gastric acid secretion.