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Ce produit n'est pas destiné à diagnostiquer, traiter, guérir ou prévenir toute maladie. Ces déclarations n'ont pas été évaluées par la Food and Drug Administration.
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Ces informations sont fournies à titre éducatif uniquement et ne remplacent pas un avis médical professionnel, un diagnostic ou un traitement. Consultez toujours votre professionnel de santé avant d'utiliser des plantes, surtout si vous êtes enceinte, allaitez, prenez des médicaments ou avez une condition médicale.
Isatis indigotica
TCM pigment herb used topically and internally for inflammatory conditions.
Qing Dai (Isatis indigotica) is a traditional Chinese medicinal pigment derived from indigo plants, primarily used for its anti-inflammatory and anti-psoriatic properties. Its key active compounds include indirubin, indigo, tryptanthrin, and isatin, which contribute to its therapeutic effects. It is applied both topically and internally for conditions such as psoriasis, mouth ulcers, fever, and mumps.
Indirubin acts as a potent inhibitor of cyclin-dependent kinases (CDKs) and glycogen synthase kinase-3β (GSK-3β), thereby modulating cell proliferation and inflammation. Tryptanthrin inhibits cyclooxygenase-2 (COX-2) and inducible nitric oxide synthase (iNOS), reducing prostaglandin and nitric oxide production. Isatin exhibits monoamine oxidase (MAO) inhibition and anti-inflammatory effects via NF-κB pathway suppression. These mechanisms collectively contribute to its antipyretic, anti-inflammatory, and anti-psoriatic actions.
TCM pigment herb used topically and internally for inflammatory conditions.
Qing Dai (Isatis indigotica) is a traditional Chinese medicinal pigment derived from indigo plants, primarily used for its anti-inflammatory and anti-psoriatic properties. Its key active compounds include indirubin, indigo, tryptanthrin, and isatin, which contribute to its therapeutic effects. It is applied both topically and internally for conditions such as psoriasis, mouth ulcers, fever, and mumps.
Indirubin acts as a potent inhibitor of cyclin-dependent kinases (CDKs) and glycogen synthase kinase-3β (GSK-3β), thereby modulating cell proliferation and inflammation. Tryptanthrin inhibits cyclooxygenase-2 (COX-2) and inducible nitric oxide synthase (iNOS), reducing prostaglandin and nitric oxide production. Isatin exhibits monoamine oxidase (MAO) inhibition and anti-inflammatory effects via NF-κB pathway suppression. These mechanisms collectively contribute to its antipyretic, anti-inflammatory, and anti-psoriatic actions.