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Ce produit n'est pas destiné à diagnostiquer, traiter, guérir ou prévenir toute maladie. Ces déclarations n'ont pas été évaluées par la Food and Drug Administration.
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Ces informations sont fournies à titre éducatif uniquement et ne remplacent pas un avis médical professionnel, un diagnostic ou un traitement. Consultez toujours votre professionnel de santé avant d'utiliser des plantes, surtout si vous êtes enceinte, allaitez, prenez des médicaments ou avez une condition médicale.
Tephrosia purpurea
Pan-tropical herb; roots used in Ayurveda for liver, spleen, and respiratory conditions.
Tephrosia purpurea is a pan-tropical herb whose roots are traditionally used in Ayurveda for liver, spleen, and respiratory conditions. Modern research indicates hepatoprotective, anti-inflammatory, bronchodilator, and antitumor activities, attributed to flavonoids, tephrosin, and other compounds. However, the presence of rotenone necessitates caution due to potential toxicity.
Hepatoprotective effects are mediated by flavonoid antioxidants that scavenge free radicals and inhibit lipid peroxidation, while tephrosin and other rotenoids exhibit anti-inflammatory activity by suppressing COX-2 and NF-κB pathways. Bronchodilation may result from inhibition of phosphodiesterase or antagonism of histamine receptors. Antitumor actions involve induction of apoptosis via mitochondrial pathways and inhibition of topoisomerase II.
Pan-tropical herb; roots used in Ayurveda for liver, spleen, and respiratory conditions.
Tephrosia purpurea is a pan-tropical herb whose roots are traditionally used in Ayurveda for liver, spleen, and respiratory conditions. Modern research indicates hepatoprotective, anti-inflammatory, bronchodilator, and antitumor activities, attributed to flavonoids, tephrosin, and other compounds. However, the presence of rotenone necessitates caution due to potential toxicity.
Hepatoprotective effects are mediated by flavonoid antioxidants that scavenge free radicals and inhibit lipid peroxidation, while tephrosin and other rotenoids exhibit anti-inflammatory activity by suppressing COX-2 and NF-κB pathways. Bronchodilation may result from inhibition of phosphodiesterase or antagonism of histamine receptors. Antitumor actions involve induction of apoptosis via mitochondrial pathways and inhibition of topoisomerase II.