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Physalis ixocarpa
The papery husks of tomatillos used in Mexican folk medicine for cancer support and diabetes.
Tomatillo husk (Physalis ixocarpa) is a traditional Mexican remedy prepared from the papery husks of the tomatillo fruit, used primarily for its hypoglycemic, anti-tumor, and antipyretic properties. Key active compounds include physalins, withanolides, fatty acids, and flavonoids, which contribute to its therapeutic effects. Evidence is limited (Grade C) but supports its traditional applications in diabetes, cancer support, and fever reduction.
Physalins and withanolides are steroidal lactones that exhibit anti-inflammatory and immunomodulatory effects, potentially through inhibition of NF-κB and STAT3 signaling pathways. Hypoglycemic effects may involve enhanced insulin sensitivity and inhibition of α-glucosidase enzyme activity. Anti-tumor activity is attributed to induction of apoptosis via caspase activation and cell cycle arrest at G2/M phase. Antipyretic effects are likely due to suppression of prostaglandin synthesis via COX-2 inhibition.
The papery husks of tomatillos used in Mexican folk medicine for cancer support and diabetes.
Tomatillo husk (Physalis ixocarpa) is a traditional Mexican remedy prepared from the papery husks of the tomatillo fruit, used primarily for its hypoglycemic, anti-tumor, and antipyretic properties. Key active compounds include physalins, withanolides, fatty acids, and flavonoids, which contribute to its therapeutic effects. Evidence is limited (Grade C) but supports its traditional applications in diabetes, cancer support, and fever reduction.
Physalins and withanolides are steroidal lactones that exhibit anti-inflammatory and immunomodulatory effects, potentially through inhibition of NF-κB and STAT3 signaling pathways. Hypoglycemic effects may involve enhanced insulin sensitivity and inhibition of α-glucosidase enzyme activity. Anti-tumor activity is attributed to induction of apoptosis via caspase activation and cell cycle arrest at G2/M phase. Antipyretic effects are likely due to suppression of prostaglandin synthesis via COX-2 inhibition.