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Ce produit n'est pas destiné à diagnostiquer, traiter, guérir ou prévenir toute maladie. Ces déclarations n'ont pas été évaluées par la Food and Drug Administration.
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Ces informations sont fournies à titre éducatif uniquement et ne remplacent pas un avis médical professionnel, un diagnostic ou un traitement. Consultez toujours votre professionnel de santé avant d'utiliser des plantes, surtout si vous êtes enceinte, allaitez, prenez des médicaments ou avez une condition médicale.
Epimedium koreanum
Perennial of East Asian forests; leaf used in TCM as kidney yang tonic and aphrodisiac — extensively researched for bone health and sexual function.
Epimedium koreanum (Yin Yang Huo) is a traditional Chinese kidney yang tonic and aphrodisiac, now extensively researched for its primary active compound icariin, a PDE-5 inhibitor that enhances nitric oxide signaling. Modern evidence supports its use for sexual dysfunction, bone health, and anti-inflammatory effects, with additional estrogenic activity from its flavonoid constituents.
Icariin and its metabolites (e.g., icariside II) inhibit phosphodiesterase-5 (PDE-5), increasing cGMP levels and enhancing nitric oxide-mediated vasodilation, which underlies its aphrodisiac effects. In bone, icariin promotes osteoblast differentiation via BMP-2 and Wnt/β-catenin pathways, while suppressing osteoclast activity through RANKL inhibition. The flavonoids also exhibit phytoestrogenic activity by binding to estrogen receptors (ERα/ERβ) and modulate inflammatory cytokines (TNF-α, IL-6) via NF-κB pathway inhibition.
Perennial of East Asian forests; leaf used in TCM as kidney yang tonic and aphrodisiac — extensively researched for bone health and sexual function.
Epimedium koreanum (Yin Yang Huo) is a traditional Chinese kidney yang tonic and aphrodisiac, now extensively researched for its primary active compound icariin, a PDE-5 inhibitor that enhances nitric oxide signaling. Modern evidence supports its use for sexual dysfunction, bone health, and anti-inflammatory effects, with additional estrogenic activity from its flavonoid constituents.
Icariin and its metabolites (e.g., icariside II) inhibit phosphodiesterase-5 (PDE-5), increasing cGMP levels and enhancing nitric oxide-mediated vasodilation, which underlies its aphrodisiac effects. In bone, icariin promotes osteoblast differentiation via BMP-2 and Wnt/β-catenin pathways, while suppressing osteoclast activity through RANKL inhibition. The flavonoids also exhibit phytoestrogenic activity by binding to estrogen receptors (ERα/ERβ) and modulate inflammatory cytokines (TNF-α, IL-6) via NF-κB pathway inhibition.