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Ce produit n'est pas destiné à diagnostiquer, traiter, guérir ou prévenir toute maladie. Ces déclarations n'ont pas été évaluées par la Food and Drug Administration.
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Ces informations sont fournies à titre éducatif uniquement et ne remplacent pas un avis médical professionnel, un diagnostic ou un traitement. Consultez toujours votre professionnel de santé avant d'utiliser des plantes, surtout si vous êtes enceinte, allaitez, prenez des médicaments ou avez une condition médicale.
Metasequoia glyptostroboides
A Chinese living fossil conifer with antioxidant properties used in folk medicine for anti-inflammatory conditions.
Metasequoia glyptostroboides, a living fossil conifer native to China, is traditionally used as an anti-inflammatory and antioxidant tonic. Modern research (evidence level C) supports its antioxidant and anti-inflammatory activities, attributed to flavonoids, tannins, and terpenes. It is typically consumed as a 2g dried leaf tea.
The antioxidant activity is mediated by flavonoid and tannin compounds that scavenge free radicals and upregulate endogenous antioxidant enzymes such as superoxide dismutase and glutathione peroxidase. Anti-inflammatory effects involve inhibition of cyclooxygenase-2 (COX-2) and lipoxygenase pathways, reducing prostaglandin and leukotriene synthesis. Terpenes may modulate nuclear factor-kappa B (NF-κB) signaling, decreasing pro-inflammatory cytokine release.
A Chinese living fossil conifer with antioxidant properties used in folk medicine for anti-inflammatory conditions.
Metasequoia glyptostroboides, a living fossil conifer native to China, is traditionally used as an anti-inflammatory and antioxidant tonic. Modern research (evidence level C) supports its antioxidant and anti-inflammatory activities, attributed to flavonoids, tannins, and terpenes. It is typically consumed as a 2g dried leaf tea.
The antioxidant activity is mediated by flavonoid and tannin compounds that scavenge free radicals and upregulate endogenous antioxidant enzymes such as superoxide dismutase and glutathione peroxidase. Anti-inflammatory effects involve inhibition of cyclooxygenase-2 (COX-2) and lipoxygenase pathways, reducing prostaglandin and leukotriene synthesis. Terpenes may modulate nuclear factor-kappa B (NF-κB) signaling, decreasing pro-inflammatory cytokine release.