Fiche d'information compilée à partir de PubMed
Cette fiche a été compilée à partir de résumés PubMed (NIH) avec l'assistance d'une IA. Chaque affirmation factuelle est citée par un article PubMed réel (voir la liste des sources). Elle n'a pas encore été révisée par un humain — confirmez auprès d'un professionnel de santé avant utilisation.
Compilé à partir de 30 articles PubMed · modèle : gemma4:31b
Résumé
Contexte
Usages traditionnels
Composés actifs
Mécanisme d'action
Preuves cliniques
In high-fat fed mice, seed oil significantly decreased low-density lipoprotein (LDL), total cholesterol (TC), and triglycerides (TG), while increasing high-density lipoprotein (HDL) [PMID:35745155]
In rats with CCl4-induced oxidative damage, tea seed oil lowered serum hepatic enzyme markers (ALT, AST, LDH), inhibited fatty degeneration, and reduced malondialdehyde [PMID:17188414]
Tea seed oil exhibited inhibitory activity on melanogenesis by inhibiting tyrosinase [PMID:29071231]
Specific sapogenins (camelliagnin A and a novel metabolite a2) demonstrated significant inhibitory effects on LPS and HMGB1-induced inflammation [PMID:38553195]
Synthèse des preuves
Sources PubMed
- 1.PMID: 40540300 (2026) — Camellia oleifera oil: unveiling health benefits and exploring novel applications. · Critical reviews in food science and nutrition
- 2.PMID: 35685348 (2022) — Applications of Chinese Camellia oleifera and its By-Products: A Review. · Frontiers in chemistry
- 3.PMID: 28793800 (2017) — Anti-inflammatory and antioxidative effects of Camellia oleifera Abel components. · Future medicinal chemistry
- 4.PMID: 17188414 (2007) — Hepatoprotection of tea seed oil (Camellia oleifera Abel.) against CCl4-induced oxidative damage in rats. · Food and chemical toxicology : an international journal published for the British Industrial Biological Research Association
- 5.PMID: 26598814 (2015)