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Ce produit n'est pas destiné à diagnostiquer, traiter, guérir ou prévenir toute maladie. Ces déclarations n'ont pas été évaluées par la Food and Drug Administration.
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Ces informations sont fournies à titre éducatif uniquement et ne remplacent pas un avis médical professionnel, un diagnostic ou un traitement. Consultez toujours votre professionnel de santé avant d'utiliser des plantes, surtout si vous êtes enceinte, allaitez, prenez des médicaments ou avez une condition médicale.
Clerodendrum serratum
Arbuste ayurvédique utilisé pour les affections respiratoires, y compris l’asthme, la bronchite et la fièvre.
Clerodendrum serratum is an Ayurvedic shrub traditionally used for respiratory conditions such as asthma, bronchitis, and fever. Modern research indicates bronchodilatory, anti-inflammatory, and antipyretic properties, attributed to compounds like clerodendrin, flavonoids, and iridoid glycosides. Evidence level is C, indicating limited clinical studies.
The bronchodilatory effects are mediated through beta-2 adrenergic receptor agonism and inhibition of phosphodiesterase, leading to smooth muscle relaxation. Anti-inflammatory actions involve suppression of pro-inflammatory cytokines (e.g., TNF-α, IL-6) and inhibition of cyclooxygenase and lipoxygenase pathways. Antipyretic effects are likely due to central prostaglandin synthesis inhibition. The iridoid glycosides and clerodendrin are thought to be the primary active constituents.
Arbuste ayurvédique utilisé pour les affections respiratoires, y compris l’asthme, la bronchite et la fièvre.
Clerodendrum serratum is an Ayurvedic shrub traditionally used for respiratory conditions such as asthma, bronchitis, and fever. Modern research indicates bronchodilatory, anti-inflammatory, and antipyretic properties, attributed to compounds like clerodendrin, flavonoids, and iridoid glycosides. Evidence level is C, indicating limited clinical studies.
The bronchodilatory effects are mediated through beta-2 adrenergic receptor agonism and inhibition of phosphodiesterase, leading to smooth muscle relaxation. Anti-inflammatory actions involve suppression of pro-inflammatory cytokines (e.g., TNF-α, IL-6) and inhibition of cyclooxygenase and lipoxygenase pathways. Antipyretic effects are likely due to central prostaglandin synthesis inhibition. The iridoid glycosides and clerodendrin are thought to be the primary active constituents.